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不知道为什么又被封禁了，我这次已经详细标注来源，甚至也翻译了，结果还是被封禁，也同样是被User:Mys_721tx，Mys_721tx管理员也都不愿意对话，就直接封禁我了，上次我被封禁，我也附上相当可靠的资料来证实我没有增加不实资料了，结果这次又封禁我增加不实资料，若认为我提供的资料有问题，请回应阿，都不正面回应，一直使用管理员的权限，而且我被封禁时间也没有进行任何编辑，Mys_721tx就封禁我，也没有询问过我是否有进行编辑，这样是正确的吗? 若认为我添加不实资料，请回应我好吗，也请先对话，不要我讲了都不回应，然后直接用权限封禁我，我个人觉得，若管理员对于资料有不实，应该举出相关文献来反驳，而不是直接封禁使用者，我觉得维基百科如果要封禁使用者，必须管理员先举出相关文献反驳，在讨论页讨论，你觉得资料哪里不实要讲啊，你也要提出相关文献阿，上次他封禁我也反应出我提供的文献讨论，结果Mys_721tx也没有回应，反而找其他理由封禁我，说我在封禁期间还进行编辑，可是我并没有进行编辑阿，Mys_721tx就是一直想封禁我，我不知道为什么他不想让我编辑条目，也不知道为什么他不愿意正面讨论，如果认为我增加的内容不合管理员的胃口，也不能这样处理阿，若认为我增加不实资料拜托你先举出相关文献反驳阿，不是都不讨论，就封禁，这样到底怎么编辑条目? 这样怎么进行条目的内容修改? 我要求Mys_721tx管理员提出可靠文献反驳，Mys_721tx不讨论不提供文献资料想封禁就封禁，那到底要怎么编辑这条目? 管理员也该提出相关文献反驳阿WW（留言）

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千村狐兔（留言） 2020年4月12日 (日) 13:09 (UTC)[回复]
处理人︰请签署

我没有增加不实资料，上述申诉于1/12就申诉了，为什么管理员没有回复? WW（留言） 2020年2月3日 (一) 02:25 (UTC)[回复]

吸毒比人类交配时更愉悦^{[来源请求]}，多巴胺增多意味着快感增多？--john doe 120@霍格华兹膜法学院^时间魔法 2020年2月3日 (一) 03:44 (UTC)[回复]

是的，多巴胺从中脑边缘途径进入NAcc壳的神经传递程度与愉悦感奖励的激励显著性高度相关，The degree of dopamine neurotransmission into the NAcc shell from the mesolimbic pathway is highly correlated with the magnitude of incentive salience for rewarding stimuli。^[1]WW（留言）

^ Berridge KC (April 2012). "From prediction error to incentive salience: mesolimbic computation of reward motivation". Eur. J. Neurosci. 35 (7): 1124–1143. doi:10.1111/j.1460-9568.2012.07990.x. PMC 3325516. PMID 22487042. "Here I discuss how mesocorticolimbic mechanisms generate the motivation component of incentive salience. Incentive salience takes Pavlovian learning and memory as one input and as an equally important input takes neurobiological state factors (e.g. drug states, appetite states, satiety states) that can vary independently of learning. Neurobiological state changes can produce unlearned fluctuations or even reversals in the ability of a previously learned reward cue to trigger motivation. Such fluctuations in cue-triggered motivation can dramatically depart from all previously learned values about the associated reward outcome. ... Associative learning and prediction are important contributors to motivation for rewards. Learning gives incentive value to arbitrary cues such as a Pavlovian conditioned stimulus (CS) that is associated with a reward (unconditioned stimulus or UCS). Learned cues for reward are often potent triggers of desires. For example, learned cues can trigger normal appetites in everyone, and can sometimes trigger compulsive urges and relapse in addicts. Cue-triggered 'wanting’ for the UCS A brief CS encounter (or brief UCS encounter) often primes a pulse of elevated motivation to obtain and consume more reward UCS. This is a signature feature of incentive salience. Cue as attractive motivational magnets When a Pavlovian CS+ is attributed with incentive salience it not only triggers 'wanting’ for its UCS, but often the cue itself becomes highly attractive – even to an irrational degree. This cue attraction is another signature feature of incentive salience ... Two recognizable features of incentive salience are often visible that can be used in neuroscience experiments: (i) UCS-directed 'wanting’ – CS-triggered pulses of intensified 'wanting’ for the UCS reward; and (ii) CS-directed 'wanting’ – motivated attraction to the Pavlovian cue, which makes the arbitrary CS stimulus into a motivational magnet."

[1] Berridge KC (April 2012). "From prediction error to incentive salience: mesolimbic computation of reward motivation". Eur. J. Neurosci. 35 (7): 1124–1143. doi:10.1111/j.1460-9568.2012.07990.x. PMC 3325516. PMID 22487042. "Here I discuss how mesocorticolimbic mechanisms generate the motivation component of incentive salience. Incentive salience takes Pavlovian learning and memory as one input and as an equally important input takes neurobiological state factors (e.g. drug states, appetite states, satiety states) that can vary independently of learning. Neurobiological state changes can produce unlearned fluctuations or even reversals in the ability of a previously learned reward cue to trigger motivation. Such fluctuations in cue-triggered motivation can dramatically depart from all previously learned values about the associated reward outcome. ... Associative learning and prediction are important contributors to motivation for rewards. Learning gives incentive value to arbitrary cues such as a Pavlovian conditioned stimulus (CS) that is associated with a reward (unconditioned stimulus or UCS). Learned cues for reward are often potent triggers of desires. For example, learned cues can trigger normal appetites in everyone, and can sometimes trigger compulsive urges and relapse in addicts. Cue-triggered 'wanting’ for the UCS A brief CS encounter (or brief UCS encounter) often primes a pulse of elevated motivation to obtain and consume more reward UCS. This is a signature feature of incentive salience. Cue as attractive motivational magnets When a Pavlovian CS+ is attributed with incentive salience it not only triggers 'wanting’ for its UCS, but often the cue itself becomes highly attractive – even to an irrational degree. This cue attraction is another signature feature of incentive salience ... Two recognizable features of incentive salience are often visible that can be used in neuroscience experiments: (i) UCS-directed 'wanting’ – CS-triggered pulses of intensified 'wanting’ for the UCS reward; and (ii) CS-directed 'wanting’ – motivated attraction to the Pavlovian cue, which makes the arbitrary CS stimulus into a motivational magnet."

[1]